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Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

A girl with a rare skin condition improved after one month of treatment with acitretin.

Cyproterone acetate is used to treat conditions like prostate cancer, early puberty, excessive sexual drive, and female androgenization by blocking male sex hormones.

A man with myotonic dystrophy type 1 had 28 skin cancers, suggesting a link between the disease and skin cancer, emphasizing the need for sun protection and regular skin checks.

CD10 helps distinguish between basal cell carcinoma and benign hair follicle tumors.

Early recognition and a team approach are crucial for managing diffuse alveolar hemorrhage in antiphospholipid syndrome.

Upadacitinib effectively treated a patient with multiple inflammatory conditions.

Arg1+ macrophages may play a role in Alopecia Areata, offering new treatment targets.

Researchers mapped and categorized specific keratin-associated protein genes on human chromosome 21q22.1.

Activating PKCα in skin causes cell death and inflammation through different pathways.

The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

ADCs can effectively treat breast cancer but may cause side effects like nausea and hair loss.

Diphenylcyclopropenone is more effective and has fewer side effects than dinitrochlorobenzene for treating alopecia areata.

PDGF-D boosts stem cell growth and movement, enhancing hair regeneration.

Upadacitinib effectively improved severe atopic dermatitis and alopecia universalis in a 29-year-old man.

A new mutation in the DCAF17 gene was found to cause Woodhouse-Sakati syndrome in a large family.

Certain skin proteins can form anchoring structures without the protein AMACO.

Congenital alopecia areata may have genetic links and topical corticosteroids are an effective treatment.

Deleting MED1 in skin cells causes hair loss and skin changes.

Upadacitinib significantly improved alopecia universalis and psoriatic arthritis in a patient.

Targeting the AhR pathway may help treat alopecia areata.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.

Longer anogenital distance may indicate a higher chance of having polycystic ovary syndrome, and measuring this distance along with hormone levels could improve diagnosis.

A new genetic mutation in the CAST gene may link PLACK syndrome to alopecia areata.

Clonally expanded CD8+ T cells cause alopecia areata.

CCC1 is crucial for pH balance in plant cells, affecting growth and stress tolerance.

Diagnosing and treating CCCA requires understanding multiple causes and using various diagnostic tools.

Certain gene variations increase the risk of alopecia areata in Koreans.

Different types of PPARγ are found in varying amounts in human skin and its parts, which could affect how skin treatments work.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.