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The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Controlling Tslp can improve health in AEC syndrome patients.

The study found that a key immune pathway protecting hair follicles is reduced in a mouse model of scarring hair loss.

AP-1 and TGFß work together to drive resistance in basal cell carcinoma, suggesting new treatment options.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

New mouse models help study melanocytic cells for melanoma research.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.

Mouse models are essential for studying and improving genetic traits in agriculture.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

The ACTH/MC2R system is crucial for controlling hair growth cycles in mice.

BMI1 is essential for preventing hair greying and maintaining hair color.

HtrA2 activity is crucial for normal hair growth by regulating fat cell development.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Alopecia areata involves specific immune cells, offering potential treatment targets.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.

AMPK activation may reduce melanoma risk in red-haired individuals.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

Researchers found that certain miRNAs, which affect immune system regulation, are differently expressed in mice with a hair loss condition compared to healthy mice.

New imaging technology can show up to 40 different markers in hair loss tissue, helping to understand hair disease better.

Bim and Puma proteins are found in developing mouse hair follicles and are involved in more than just cell death.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

AUC and APL are distinct conditions needing careful clinical assessment.

Pmg-1 and Pmg-2 are new genes important for skin and mammary gland development.