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Dopaminergic neurons in the gut have distinct subtypes, some releasing both dopamine and acetylcholine.

Short-term diabetes can cause changes in brain steroids, cholesterol balance, and mitochondrial function.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Mitochondrial stress can lead to atopic dermatitis.

Gut microbes significantly affect brain steroid levels.

Immature hair cells can grow and change into different types of hair cells over time.

Male tissue has more cell death than female tissue after ischemia, and some neurosteroids only protect female cells.

The study found no significant difference in stress hormone levels between people with alopecia areata and healthy individuals, suggesting that the disease is not caused by an overactive stress response system.

Localized ROS production is essential for cell growth and movement in plants and animals.

Increasing TSPO in the brain may help improve memory problems.

Neurosteroids play a key role in controlling the brain-adrenal gland activity in pregnant sheep, both in normal and stressful situations.

Skin and hair cells release serotonin and histamine naturally, which could help improve skin health.

Progesterone protects neurons from damage by converting to allopregnanolone, which works through GABAA receptors.

The spiny mouse regenerates ear tissue asymmetrically, with gene expression differences possibly explaining its unique healing abilities.

Arg1+ macrophages may play a role in causing alopecia areata.

Certain proteins and their receptors are more active during the growth phase of human hair and could be targeted to treat hair disorders.

Glioblastoma cells can make androgens, which might help the tumor grow.

Low-oxygen conditions and ECM degradation products increase the healing abilities of perivascular stem cells.

Children with autoimmune gastritis showed improved intestinal health over time and should be checked for other autoimmune conditions.

MRI can show unusual brain changes in adrenomyeloneuropathy.

Early-life neurosteroid changes affect adolescent exploration and adult behavior.

Human dermal fibroblast proteins help restore nerves during healing.

NGF-modified hair follicle stem cells may help treat Alzheimer's by improving brain function and reducing harmful proteins.

Certain substances can help skin cells become anti-inflammatory, aiding in tissue repair.

γδ T cells help control tissue scarring and blood vessel growth in response to foreign objects.

Altered metabolism can help control seizures by changing brain signaling and energy use, suggesting new treatments for epilepsy.

Genetically repaired stem cells may treat certain genetic diseases, Th17 cells are key in fighting systemic fungal infections, hair loss in AGA is due to progenitor cell loss, and α-synuclein transfer might contribute to Parkinson's disease progression.