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An 8-year-old girl developed a rare skin condition in a linear pattern on one side of her body after a lung infection, which improved with treatment.

A woman had a rare skin condition with recurring painful nodules that heal in 6 weeks, often without needing treatment.

Scarring alopecia involves increased immune cells and specific gene changes near damaged hair follicles.

People with alopecia areata have fewer IL-10 producing immune cells, which might contribute to the condition.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

Alopecia areata is an autoimmune disease causing hair loss, and targeting macrophages may help treat it.

Alopecia areata patients have fewer protective regulatory B cells, which may contribute to the disease.

In alopecia areata, certain immune cells increase and express a protein linked to immune activation.

Trichohyalin in hair can trigger immune attacks in alopecia areata.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

Natural killer cells play a key role in autoimmune skin diseases like pemphigus vulgaris.

Expanding regulatory T cells may help treat alopecia areata by reducing harmful immune cells.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Both induced and spontaneous AA lymphocytes can cause alopecia areata in mice.

Severe alopecia areata involves higher levels of certain immune cells, which can be normalized with betamethasone.

Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

CD3+ T-cell presence is a reliable marker to tell apart alopecia areata from pattern hair loss.

The patient had a severe allergic reaction to gold treatment.

Melanocytes might be targeted by the immune system in people with alopecia areata, but more research is needed.

Alopecia areata can be an early sign of Hodgkin’s lymphoma and may improve with lymphoma treatment.

Targeting the AhR pathway may help treat alopecia areata.

A man got six skin cancers within a year after a cell transplant for leukemia but was cancer-free 32 months later; skin checks are important post-transplant.

Alopecia areata involves specific T-cells, unlike androgenetic alopecia.

AI can help diagnose Follicular Lymphoma by accurately identifying specific cell types.

PCFCL may have unrecognized subtypes and needs more research.