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Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

Researchers found that hair follicle stem cells can become squamous cell carcinoma due to Ras activation, which could lead to new treatments.

The research found a way to identify and study skin cells with stem cell traits, revealing they behave differently in culture and questioning current stemness assessment methods.

Some aggressive scalp cancers are hard to treat and can be deadly, even when removed with specialized surgery.

Stem cells and their surrounding environment in hair follicles work closely together, affecting hair growth and having implications for cancer and tissue regeneration.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

High lactate dehydrogenase activity is not necessary for the growth of squamous cell carcinoma.

ETS2 is crucial in squamous cell carcinoma development and could be a therapeutic target.

ETS2 drives cancer progression in squamous cell carcinoma and is linked to poor patient outcomes.

Keratin 15 helps maintain skin cell growth and repair.

Long-standing benign tumors can become cancerous, especially in people with weakened immune systems.

GLI1 might protect against the start of skin cancer and is not linked to cancer severity.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Photodynamic therapy successfully treated skin cancer in a 53-year-old woman.

Lower SMAD2/3 activation predicts more severe skin cancer.

Skin cancer in sun-exposed areas is more likely to come back.

Treatment restored normal sexual characteristics and blood condition in a patient with testicular cancer.

Cancer-associated fibroblasts promote tumor growth in skin cancer.

Increased methylation of the Filip1l gene may contribute to aggressive skin cancer.

A man with a rare scalp cancer was successfully treated, highlighting the need for early management to prevent spread and complications.

Targeted radionuclide therapy shows promise for improving head and neck cancer treatment but needs more research.

High DKK1 levels predict worse survival in head and neck cancer.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

HPV16-transformed cells can change human skin cell properties, aiding tumor growth.

Transplant patients on immunosuppressive medications have a higher risk of skin cancer, and managing this involves balancing medication with cancer risk.

Diuretics can cause serious side effects and should be used carefully.

Matriptase is crucial for skin barrier, hair growth, and may contribute to skin cancer.

ΔNp63α stops TAp73β from working in skin cancer by blocking its access to specific genes, not by directly interacting with it.