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Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Blocking BACE1 and BACE2 enzymes causes hair color loss in mice.

Mice with CBS deficiency are healthier on a low-methionine diet.

Mice with the ab gene have abnormal sebaceous gland development, affecting sebum production.

A cat's poor wound healing was linked to a genetic deletion in the COL5A1 gene.

Targeting ornithine decarboxylase can help prevent skin cancer.

Alopecia areata in these mice is inherited, more common in young females, and can be treated with triamcinolone acetonide.

Mutations in Far2 mice cause hair loss due to sebaceous gland issues.

Overexpression of E2F1 can lead to skin tumors and disrupt hair growth.

The C3H/HeJ mouse model is useful for studying and testing treatments for alopecia areata.

A new mouse mutation causes skin and hair issues, influenced by another gene.

Parental uveitis increases offspring's risk and severity of autoimmune eye disease.

A new mutation in the PLEC gene causes a rare condition with skin blistering, muscle weakness, and hair loss.

Msx2 deficiency in mice leads to bone growth and organ development problems.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

Trichloroethylene causes skin inflammation in mice by increasing certain immune proteins.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Mice can regenerate ear tissue without the p53 protein.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Turning off the Lhx2 gene in mouse embryos leads to slower wound healing and scars.

FoxN1 overexpression in young mice harms immune cell and skin development.

Multiple pregnancies prevent skin disease but worsen kidney disease in certain mice.

HPV genes in mice improve ear tissue healing by speeding up skin growth and repair.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

MPA increases cancer spread by boosting Eph A2 activity.

Overexpressing COX-2 in mice skin reduces skin tumor development.