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Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

Basal cell carcinomas need extra mutations to grow from small to large tumors.

Lymphoid-specific helicase (Lsh) is crucial for skin growth, change, and healing after injury.

PBMCsec can help reduce and improve thick skin scars.

Collagen XVII and CD151 affect cell movement, with CD151 inhibiting migration when bound to integrins.

Localized dystrophic epidermolysis bullosa (DEB) can lead to scarring alopecia, highlighting the need to recognize and address this complication.

Skin cell clumping for hair growth is improved by a protein called fibronectin, which helps cells stick and move better.

GLABRA2 represses root hair formation by inhibiting a specific gene.

IL10_ApoEVs improve skin healing and reduce scarring.

Deer antler stem cell fluid helps regenerate tissue better than fat-derived stem cell fluid.

The model helps understand scar contraction and develop new treatments.

Certain proteins help nerve cells branch, and other findings relate to cancer, stem cell behavior, and cell division.

β-catenin, Shh, and Bmp signaling control hair follicle development.

Ultrasound-activated gel with stem cell vesicles improves skin healing and regeneration.

Dermal EZH2 controls skin cell growth and differentiation in mice.

A virus protein can activate a pathway that may lead to abnormal hair follicle development.

Abnormal ECM and immune cell interactions can cause skin diseases.

LPA3 signaling in the uterus is crucial for placental formation and fetal development.

Blocking cell death in hair follicles can lead to impaired hair growth.

CMG2 and TEM8 receptors have distinct roles in skin and growth disorders, affecting collagen breakdown and growth.

New mouse models of Pemphigus show severe symptoms and need better treatments.

Neutrophils quickly respond to skin injury.

The Ovol2-Zeb1 circuit is crucial for skin healing and hair growth by guiding cell movement and growth.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The biofilm enhances skin healing by promoting cell growth and blood vessel formation.

Using a specific binding agent and low doses of FK506 can stimulate stem cells, speeding up skin healing by 25% and improving skin quality in rats and mice.

FGF-9 speeds up the early development of certain organs, showing potential for organ regeneration.