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Alloxan diabetes, methylthiouracil, cortisone, and adrenaline affect how white mice hair follicles use glucose and cystine and their cell division.

Hairless mice lose hair by 3-4 weeks, develop thicker, folded skin, and show pigmentation differences.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

The ACTH/MC2R system is crucial for controlling hair growth cycles in mice.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

B6.Cg-Tyr c−2J Hr hr /J mice have a stronger delayed sunburn reaction and are good for UV research.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

The B2C promoter works in sheep cells but not in mouse embryos.

Mice with extra sheep genes had hair that fell out and regrew in cycles.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

K6/ODC transgenic mice are effective for quickly identifying cancer-causing chemicals.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

A mouse model was created to study hair loss similar to humans.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Knockout mice showed anemia and hair loss, suggesting other ways exist for iron absorption.

A new mouse mutation causes skin and hair issues, influenced by another gene.

A new genetic mutation in mice causes permanent hair loss and skin wrinkling.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

The Naked gene in mice causes abnormal sebaceous glands and disrupts hair follicle organization.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

A zinc-deficient diet stunted growth and affected organs in mice, with C57BL/6J mice showing more severe symptoms.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

The lanceolate hair-J mutation in mice helps understand human hair disorders like Netherton's syndrome.

The improved genome of the African spiny mouse helps study its tissue regeneration.