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Overexpressing PKCepsilon in mice reduces papillomas but increases carcinomas.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Mice with human chymase had a higher death rate when exposed to a toxin compared to normal mice.

The hr gene is crucial for skin and hair health, with mutations causing hair disorders.

Scube3 gene affects mouse embryo growth in multiple areas, but needs more research.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Mice without Vitamin D receptors have hair growth problems because of issues in the hedgehog signaling pathway.

A new mouse mutation causes skin and hair defects due to a gene change.

The Itpr3 gene causes a specific hair pattern in mice.

Progeroid mouse models show signs of early aging similar to humans, helping us understand aging better.

Noggin gene inactivation causes skeletal defects in mice, varying by genetic background.

Adult spiny mice recover better from heart attacks than common lab mice.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

ARHGEF3 is essential for proper hair follicle development in mice.

EGCG promotes hair growth in mice.

Mouse Epidermal Neural Crest Stem Cells can become various cell types.

Encapsulating cell-free fat extract in GelMA hydrogel improves skin flap survival in mice.

Careful selection of mice by genetics and age, and controlled housing conditions improve the reliability of hair regrowth in wound healing tests.

High doses of TCB cause severe health issues in marmoset monkeys.

Certain substances can decrease or increase exploratory behavior in rodents.

Lack of functional CYLD in mice leads to early aging and cancer.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.