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Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

Canine hair follicle cells show stem cell properties, aiding hair growth.

A specific gene mutation causes a severe skin disorder in a family.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

Immunomodulatory therapies are effective for treating cutaneous lymphoma, particularly in early stages.

Blocking IL-17 can reduce skin inflammation in a mouse model of pityriasis rubra pilaris.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

Touch dome keratinocytes in adult skin have traits of different skin cell types.

FCER1A and RGS1 may help diagnose and treat systemic lupus erythematosus.

Daclizumab may cause psoriasis-like skin problems in multiple sclerosis patients.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Adipose-derived stem cells help heal radiation skin damage by reducing cell death and inflammation.

Targeted siRNA therapy may be a promising treatment for KID syndrome by reducing mutant gene expression and improving cell communication.

ZD1839, a cancer drug, can cause mild skin rashes that are treatable without stopping the medication.

Dysplastic nevi have unique gene expressions, making them distinct from common melanocytic nevi.

Kdm6b is crucial for skin cell differentiation.

PRO-C22 can help diagnose and monitor the severity of hidradenitis suppurativa.

IL-18 signaling helps mature Tregs move into the thymus.

A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

TPA is about 50 times more effective at promoting tumors than MZ.

Increased methylation of the Filip1l gene may contribute to aggressive skin cancer.

Activating PPAR-γ signalling can protect hair follicle stem cells from damage caused by chemotherapy.

A new TP63 mutation was found in a baby with EEC syndrome, showing the need for TREC testing to check for immune issues.

Clonally expanded CD8+ T cells cause alopecia areata.

CLK1 is needed for skin cells to become epidermal cells but not sebocytes.

High DKK1 levels predict worse survival in head and neck cancer.

Higher levels of CCL7 are linked to more severe hair loss in alopecia areata patients.

Psoriasis may be chronic because it lacks certain immune system controls that prevent overreaction.