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Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

Higher MIF levels in alopecia areata patients suggest it could be a treatment target and disease predictor.

Stromal cells in melanoma promote tumor growth and spread.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Alopecia areata patients have higher levels of certain immune receptors, suggesting new treatment possibilities.

Hair follicles help attract immune cells to minor skin injuries.

RAR-gamma 1 is important for normal skin maintenance and differentiation.

CD34 marks potential stem cells in dog hair follicles.

Kdm6b is crucial for skin cell differentiation.

CD44 variant changes start alopecia areata, but don't maintain it.

Basal cell carcinomas need extra mutations to grow from small to large tumors.

Androgen receptors found in monkey scalps, similar to humans, affect hair growth.

UTMD with diclofenac and Doxil® improves cancer treatment by boosting immune response and reducing tumor-supporting cells.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Stem cells slowed lung tumor growth but increased colon tumor growth in mice.

CD4 T cells can cause alopecia areata by activating CD8 T cells to attack hair follicles.

A new mouse model helps study melanocyte cells using GFP expression.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

Alpha 6 + /MHCI - cells have stem cell traits and are similar to mouse hair follicle stem cells.

Aging in one type of stem cell can cause aging-like changes in various organs.

TAZ boosts fat cell formation in goat stem cells by activating a specific signaling pathway.

A specific microRNA, chi-miR-30b-5p, slows down the growth of hair-related cells by affecting the CaMKIIδ gene in cashmere goats.

Stem cells help remove dead cells to keep tissues healthy by balancing cell replacement and clearance.

Rapamycin and anti-EGFR antibody reduce LAM/TSC cell migration and blood vessel growth in the uterus.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

Hair follicle stem cells can become nerve cells using specific treatments.

Trichoblastomas may mimic fetal skin development by having many Merkel cells, unlike adult skin.

A parasite-derived molecule speeds up skin healing and affects immune cell behavior without increasing scarring.