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New hair regrowth model proposed, imiquimod found to kill skin cancer cells, T-cadherin loss linked to invasive skin cancer, no clear gene link to skin cancer after transplant, and study on children's hair loss shows male dominance and genetic ties.

New biomarkers and potential treatments for skin diseases were identified.

Higher levels of certain immune cells in hair follicles may contribute to alopecia areata.

A rare genetic deletion in the KRT1 gene causes unique skin symptoms in a family.

The trichohyalin gene is located at chromosomal region 1q21 with other skin-related protein genes.

Innate immunity genes in hair follicle stem cells might have new roles beyond traditional immune functions.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

Stem cells play a key role in nonmelanoma skin cancers, with different origins and genetic changes linked to basal and squamous cell carcinomas.

Hair follicle stem cells have greater longevity and adhesion, while transit-amplifying cells are more mobile.

Trichohyalin is found in non-hair tissues and works with filaggrin in certain skin areas and conditions.

New mutations in KRT83 and KRT86 are linked to the hair disorder monilethrix.

ATP-sensitive potassium channels are important for hair growth.

Human and bovine hair follicles have distinct cytokeratins specific to hair-forming cells.

Basal cell carcinomas have much higher levels of Vitamin D3 receptors compared to healthy skin.

The research found a way to identify and study skin cells with stem cell traits, revealing they behave differently in culture and questioning current stemness assessment methods.

K6hf is found in specific parts of hair follicles, nails, and tongue, and is linked to hair growth and structure.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Lower proximal cup cells, not bulge stem cells, regenerate hair follicles after chemotherapy.

Sciellin is a protein that helps form protective layers in skin, hair, and nails.

Melanocytes in the outer root sheath are likely stem cells that grow fast but stay immature.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

HPV16-transformed cells can change human skin cell properties, aiding tumor growth.

Memory T cells in the skin balance staying put and moving into the blood, clustering around hair follicles, and increasing in number after infection.

Nucleic acids trigger chemokine production in skin cells, affecting skin inflammation.

The cyst had unusual keratin spherules and resembled bone marrow.