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Discoid lupus erythematosus involves immune activation and fibrosis around hair follicles, with shared pathways across humans, dogs, and mice, suggesting potential treatments for both humans and animals.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Targeting Vδ1+T-cells may help treat alopecia areata.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

A specific type of immune cells, called CD301b-expressing macrophages, are crucial for skin repair processes.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

Allergic contact dermatitis may promote hair growth by activating hair follicle stem cells.

Tofacitinib helped most patients with alopecia areata regrow hair and changes in immune cells were linked to the treatment's effectiveness.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

Cytotoxic T cells cause hair loss in chronic alopecia areata.

A patient with a severe type of hair loss experienced partial hair regrowth after treatment with a specific light therapy and a medication called interferon α2a.

Thymosin beta-4 promotes hair growth in mice.

BTNL proteins help control inflammatory bowel disease by maintaining specific immune cells.

Tofacitinib and low-dose IL-2 may help maintain hair regrowth in alopecia areata without ongoing treatment.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Higher PD-1 levels are linked to fewer immune cells in hair follicles in alopecia areata.

Using regulatory T cells and Rapamycin together improves chronic graft-versus-host disease treatment outcomes in mice.

Merkel cell polyomavirus can infect and persist in skin cells, evading the immune system, but certain treatments can control it.

CD201+ fascia progenitors are essential for wound healing and could be targeted for treating skin conditions.

Hair follicles help skin immune recovery after UVB exposure.

The new ddPCR method reliably detects unwanted viruses in CAR-T cell products, ensuring their safety for patients.

Low power red laser with efavirenz can reduce HIV-1 infection to undetectable levels and improve diagnosis.

CTCL patients can safely continue treatment during COVID-19 with proper safety measures.

Suppressing eIF4E can prevent hair loss from chemotherapy.

Cell-based therapy using specific immune cells may help treat alopecia areata by promoting hair regrowth.

The assay effectively identifies compounds that affect immune cell activation.