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A specific genetic trait in tumor cells is linked to longer survival without disease in certain lymphoma patients.

Special immune cells called Tregs can help prevent lung scarring by blocking a specific growth factor.

T-cell patterns in skin help distinguish alopecia areata from androgenetic alopecia.

Tofacitinib and low-dose IL-2 may help maintain hair regrowth in alopecia areata without ongoing treatment.

Ritlecitinib may help treat alopecia areata by protecting hair follicles.

A mouse model for studying scleroderma in chronic graft-versus-host disease was successfully created.

Alopecia areata involves both innate and adaptive immunity, with specific genes linked to the disease.

PDCD4 is important for controlling skin cell growth and healing.

People with atopic dermatitis have more CD4+ T cells that respond to a certain bacterial lipid, which may play a role in the skin condition's inflammation.

Treg dysfunction is linked to various autoimmune skin diseases, and understanding Treg properties is key for new treatments.

Mouse skin cancer progression involves a unique group of cells marked by ABCG2 and MTS24.

Researchers found stem cells in dog hair follicles using specific markers.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Farudodstat can prevent hair follicle immune damage linked to alopecia areata.

Scientists found markers called CD34 and CD200 that help identify stem cells in mouse and human hair follicles.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Tissue-resident memory T cells can protect against infections and cancer but may also contribute to autoimmune diseases.

The model showed that immune system guardians and the cytokine interferon-γ are key in alopecia areata progression.

Blocking certain immune signals can reduce skin damage from radiation therapy.

IL-17C is important in inflammatory skin diseases and could be a target for treatment.

Increasing regulatory T cells may help treat alopecia areata by reducing autoimmunity and promoting hair growth.

CD34+ cells from fat tissue help form hair follicles and blood vessels in skin.

Some patients with a type of skin lymphoma can experience a rare, non-scarring hair loss that looks like another hair loss condition but has distinct features.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Low power red laser with efavirenz can reduce HIV-1 infection to undetectable levels and improve diagnosis.

Researchers found that certain miRNAs, which affect immune system regulation, are differently expressed in mice with a hair loss condition compared to healthy mice.

Scarring alopecia involves increased immune cells and specific gene changes near damaged hair follicles.

The new Hairless R/J mice model improves imaging for tumor monitoring and cancer therapy evaluation.