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CD28 is a promising target for treating alopecia areata with belatacept.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

Blocking CD44 can reduce leukocyte migration in autoimmune skin diseases.

Gamma/delta T cells help defend skin against heavy metals.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

Enhancing Tregs can protect against alopecia areata.

Bone marrow-derived cells contribute to skin tumors, suggesting new treatment targets for non-melanoma skin cancers.

Cutaneous lupus patients have higher levels of certain immune cells in their blood and skin.

B cells can both help and hinder the body's defense against melanoma.

Scientists found a new type of skin cell that could help with skin repair and these cells work better with a certain protein.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Clonally expanded CD8+ T cells cause alopecia areata.

Dual TCR Treg cells are common in mouse tissues and vary by location.

Certain human skin cells marked by CD44 and ALDH are rich in stem cells capable of long-term skin renewal.

Langerhans' cells are not involved in hair depigmentation in these mice.

Three characteristics of plasmacytoid dendritic cells help tell apart lupus-related hair loss from LPP.

T cells with memory features grow in number and gather around hair follicles when there are not enough immune cells.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Canine hair follicle cells show stem cell properties, aiding hair growth.

Certain immune cells are linked to non-scarring hair loss, suggesting potential for immune-targeted treatments.

Transcutaneous vaccination using nanoparticles can enhance immune responses and reduce basal cell carcinomas.

Alopecia areata involves specific immune cells, offering potential treatment targets.

The Fas/FasL pathway may play a role in alopecia areata.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Mouse skin has special cells in the epidermis that decrease with age and are linked to keratinocyte stem cells.

CD200 is not a reliable marker for identifying stem cells in all skin types.

Corticosteroid therapy reduces specific immune cells and promotes hair growth in alopecia areata patients.