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A rare case of a transplant patient developing a skin condition linked to HPV-49.

γδT cells can protect hair follicles from alopecia areata and promote hair regrowth.

Different types of resting melanocyte stem cells have unique characteristics and vary in their potential to become other cells.

Removing certain immune cells in mice causes their hair to enter the growth phase earlier than usual.

A method was found to select hair follicle stem cells, and beta-catenin helps them grow and change.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

Certain mature cells in mouse lungs can turn back into stem cells to aid in tissue repair.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Skin-resident memory T cells may contribute to chronic alopecia areata and baricitinib could be a potential treatment.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

LL-37 helps stem cells grow and move, aiding tissue regeneration and hair growth.

Activin increases skin tumor formation, skin Tregs help hair growth, lymph-node removal doesn't improve melanoma survival, cells can revert to stem cells in wound healing, and skin bacteria produce peptides that may treat infections.

Lower proximal cup cells, not bulge stem cells, regenerate hair follicles after chemotherapy.

RAR-gamma 1 is important for normal skin maintenance and differentiation.

γδTregs may help treat autoimmune diseases like alopecia areata by promoting hair regrowth and reducing immune attacks.

Activin B improves wound healing and hair growth by helping stem cells move using certain cell signals.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Certain genes help dermal papillae cells in hair follicles grow and group together.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Lentiviral vector effectiveness in skin is influenced by external factors, not receptor availability.

Replacing defective mesenchymal cells with normal ones fixes thymic growth issues in 22q11.2DS mouse models.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

Cells in hair follicles help create fat cells in the skin by releasing a protein called Sonic Hedgehog.

Mechanical stimuli and CCL2 can help regenerate hair follicles in adult mice.

Abnormal Hedgehog signaling in blood cancers may help tumors grow and resist chemotherapy, suggesting potential for targeted treatments.

Melanocyte stem cells can become melanoma, resembling human melanoma.

miR-22 helps skin cancer grow and spread by activating specific cell signals.

Rapamycin increased survival in mice with severe chronic graft-versus-host disease by expanding regulatory T cells.

Recognizing RSCC is crucial due to its aggressive nature and high risk of poor outcomes.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.