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PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Targeting the PGI2 pathway may help heal diabetic foot ulcers.

JMJD3 and NF-κB activate Notch1, which is essential for skin cell movement and wound healing.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

A specific RNA, circNlgn, contributes to heart damage and scarring caused by the cancer drug doxorubicin.

TLR3 activation helps improve skin and hair follicle healing in mice.

Vitamin D3-activated cell byproduct promotes hair growth in mice by increasing blood vessel growth.

Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

IL-1 signaling is crucial for hair follicle stem cell growth and wound healing.

A protein called CBP is found in prostate cancer and can increase the effectiveness of certain prostate cancer treatments.

IL-19 and IL-24 help cells respond to DNA damage and could be targeted for cancer and age-related disease treatments.

The vitamin D receptor can regulate genes and affect hair and hormone levels without its usual activator.

Activating TLR3 helps improve skin and hair follicle regeneration after wounds.

CD200- cells in hair follicles have a higher ability to regenerate hair.

Claudin expression changes help the skin respond to injury.

Double stranded RNA helps skin wounds heal by coordinating specific proteins and signaling pathways.

The study concluded that the developed models are effective for studying hair growth mechanisms and testing new treatments.

Mast cells and the CB1 receptor may be key in causing IgA vasculitis.

Activating TRPMLs helps human cells important for hair growth and increases hair growth in mice.

Targeting ATM could help manage heart cell enlargement due to pressure overload.

Inhibiting ODC can prevent UV-induced skin cancer.

Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

A specific gene change plus an additional mutation in the same gene cause hereditary trichilemmal cysts.

BMP-2 activates the Dlx3 gene in mouse skin cells, important for hair and skin development.

TCDD reduces EGF receptor activity and causes various developmental changes in animals.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.

Blocking CD44 can reduce leukocyte migration in autoimmune skin diseases.

Bacteria can help skin regenerate through a process called IL-1β signaling.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.