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Short-term treatment with ROCKi increases skin cell growth without changing stem cell features.

KRT80 may worsen cancer by increasing growth and spread, but its full effects on treatment and outcomes need more research.

Gata6 is important for protecting hair growth cells from DNA damage and keeping normal hair growth.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.

Skin cells can naturally limit the growth of cancerous changes by balancing cell renewal and differentiation.

p63 is essential for skin cell growth and differentiation by controlling specific gene networks.

Dock5 is important for skin healing and could help treat diabetic wounds.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

CXXC5 can both suppress and promote cancer, making it a complex target for treatment.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

DKK2 and SOSTDC1 together are necessary for the normal timing of the first regression phase in the hair growth cycle.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

Cancer-associated fibroblasts promote tumor growth in skin cancer.

Mouse keratin 6 isoforms have different expression patterns in various tissues.

Akt2 protein is essential for normal cell division in early mouse embryos.

Sgk3 is essential for normal hair follicle growth and maintenance.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Kdm6b is crucial for skin cell differentiation.

Akt2 and SGK3 are both important for normal hair growth and development.

Imatinib can repigment grey hair, while SU11428 can cause temporary hair depigmentation.

Hair follicle cells resist turning into skin cells.

Targeting ornithine decarboxylase can help prevent skin cancer.

BI 2536 had limited effectiveness against several advanced cancers and caused significant side effects.

PTCH gene mutations contribute to basal cell carcinoma development.

Connexin-26 gene mutations may increase cancer risk in KID syndrome patients.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Beta-catenin boosts hair follicle cell growth by increasing c-myc expression.