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Circular RNA ERCC6 helps activate stem cells important for cashmere goat hair growth by interacting with specific molecules in an m6A modification-dependent way.

The structure of SRD5A reveals how it reduces steroids, aiding drug design for related health conditions.

A new mouse mutation causes skin and hair defects due to a gene change.

Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Corneodesmosin is crucial for skin and hair health, and its dysfunction can cause skin and hair disorders.

Lack of TG2 increases fat storage and lowers cell cleanup in skin oil cells.

Targeting protein S-palmitoylation could lead to new skin disease treatments.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

Certain genetic changes in the LSS gene cause a rare skin and hair condition.

NADPH oxidase 4 is key for stem cell activity and growth under low oxygen.

The TIP1 gene is crucial for normal plant cell growth in Arabidopsis.

HAT-L4 is crucial for preventing body fluid loss by maintaining skin barrier integrity.

The compound 4c effectively inhibits the enzyme linked to hair loss.

Syndecan-1 is essential for maintaining skin fat and preventing cold stress.

VDAC2 promotes cell death in cashmere goat hair follicles through the P53 pathway.

CYP154C7 enzyme can effectively modify steroids and is promising for commercial use.

The research found enzymes in Stephania epigaea that help make cepharanthine.

Fatty acid transport protein 4 is essential for skin and hair development.

Lack of Cisd2 disrupts calcium balance in cells, leading to poorly functioning neutrophils.

Mutations in the DSG4 gene cause fragile, sparse hair in humans, mice, and rats.

Sebaceous glands help study fatty acid transporters and binding proteins.

The SYP123-VAMP727 complex is important for transporting materials that harden the root hair shank in Arabidopsis.

Cadd4 effectively reduces cholesterol levels without side effects.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Enzymes called PADIs play a key role in hair growth and loss.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

The NIPAL4 mutation c.527C>A is common in Romanian patients with autosomal recessive congenital ichthyosis.

Cis-UCA can form new compounds without enzymes, and sulphide donors reduce its UVB-induced toxicity in skin cells.