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Six new genetic regions linked to early hair loss also connect to Parkinson's disease and prostate cancer, possibly leading to new treatments.

Grizzly and black bears have different stress and hormone responses to salmon availability, influenced by nutrition and social competition.

Certain genetic variants in keratins increase the risk of tooth decay.

Adding a specific gene to skin cells can help treat skin disorders like psoriasis.

WNT10A and EBF1 interaction affects hair growth in male-pattern baldness.

MC4R gene variants not linked to female hair loss.

Blocking certain receptors in the lungs might help treat a specific type of asthma.

γδ T cells are essential for wound healing after poxvirus infection.

Removing the p75 gene in mouse skin cells didn't affect their skin or hair growth.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Adult spiny mice recover better from heart attacks than common lab mice.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Only Deomyinae rodents can regenerate complex tissues.

Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Spiny mice can regenerate tissues instead of forming scars.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

The gene causing hair loss and heart issues in rough coat mice is still unknown.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

A new mouse mutation causes skin and hair issues, influenced by another gene.

Overexpressing ovine β-catenin in mice skin increases hair follicle density and growth.

Human hair follicles have a unique way of using energy and might use the Cori cycle; blocking CCR5 could help treat hair loss.

Mouse embryos can develop in chick embryos, but they grow smaller with some organ issues.

Corneal health relies on cell migration and cooperation with the lacrimal gland, not Bmi1+ cells, and Eda gene mutations can cause dry eye disease.

Some mutant mice have hair with abnormal cross-linking, mainly in the cuticle, not affecting other hair parts.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Sebaceous lipids are crucial for keeping skin and eyes healthy in mice.