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The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

Ultrasound-assisted gene therapy could revolutionize tissue regeneration by improving gene delivery.

Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Mice without the Sept4/ARTS gene heal wounds better due to more stem cells that don't die easily.

Retinoic acid and DMSO improve gene delivery to mouse skin for potential hair and skin disease treatment.

The research suggests that a specific skin gene can be controlled by signals within and between cells and is wrongly activated in certain skin diseases.

Gene therapy shows promise for treating hair loss by targeting hair follicles.

Scientists made a mouse model of a serious skin cancer by changing skin cells with a virus and a specific gene, which is similar to the disease in humans.

Researchers found 19 genes important for root hair growth in a plant called Arabidopsis.

Smart nano-PROTACs improve cancer treatment by targeting proteins more precisely and reducing side effects.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

The environment around the time of conception can change the VTRNA2-1 gene in a way that lasts for years and may affect disease risk.

A new mouse mutation causes skin and hair defects due to a gene change.

PROTACs offer a new, precise way to treat cancer by breaking down harmful proteins.

Gene therapy shows promise for enhancing physical traits but faces ethical, safety, and regulatory challenges.

The method quickly analyzes hair growth genes and shows that blocking Smo in skin cells stops hair growth.

Mice with a specific BRCA1 mutation have a higher risk of tumors, especially in the uterus and ovaries.

RNase L suppresses regeneration in mammals.

DNA changes in tetraploid wheat improve root growth and nitrogen use.

CTCF protein is essential for skin and hair follicle development in mice.

Two specific SNPs in the TRPS1 gene cause excessive hair growth by altering the protein's structure.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

RNase L hinders hair follicle regeneration by altering immune signals.

Copper supplements during pregnancy improve survival and development in mutant mice.

Transgenic sheep cells with spider silk gene were successfully created for future sheep hair expression.

The AVET system effectively delivers genes to human keratinocytes and may help treat skin diseases.

The improved genome of the African spiny mouse will help understand its tissue regeneration abilities.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.