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Mirex seems to promote a unique group of skin cells different from those affected by another tumor promoter, TPA.

A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Chronic T. gondii infection may harm male fertility.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

CDP is crucial for lung and hair follicle cell development.

MILL molecules are unique immune proteins in mice that don't need TAP to appear on cell surfaces.

The improved genome of the African spiny mouse will help understand its tissue regeneration abilities.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

The research identified new skin traits in mice, some linked to human skin conditions.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

A mutation in mice causes hair loss and immune problems.

Mutant mice help researchers understand hair growth and related genetic factors.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

The scraggly mutation causes hair loss and skin defects in mice.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Mouse models help study genetic skin diseases.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Suppressing ODC activity reduces tumor growth in hair follicles.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.