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A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

CREB, a protein that can promote cancer traits, is controlled by β-catenin in skin cancer cells.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

C.I. Acid Orange 3 caused cancer in female rats but not in male rats or mice.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

Dual TCR Treg cells are common in mouse tissues and vary by location.

CCDC22 and CCDC93 are essential for root and root hair growth in Arabidopsis.

A mutation in the VDR gene affects hair cycling without needing ligand binding.

Diphencyprone helped most patients with alopecia areata regrow some hair.

Blocking CRF1 receptors improved male hormone levels and reduced testicular tumor size in men with a specific adrenal condition.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

A new test more accurately detects citrulline in hair follicles and pilomatrixomas.

dAR-6–1 is a promising new treatment for hair loss that works better than minoxidil.

TRIV-509 quickly improves skin barrier and cell health in atopic dermatitis.

TRα and CRABPII genes change their activity levels during goat fetal skin development.

A specific gene mutation causes a severe skin disorder in a family.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

CRABP1 boosts hair cell growth in Hu sheep by affecting key genes.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

CCCA may involve the PD1/PDL1 pathway and increased caspase 3, leading to permanent hair loss.

Certain mutations in the H6PD gene cause Cortisone Reductase Deficiency by affecting hormone production.

CRH in the skin acts like the body's stress response system, affecting cell behavior and immune activity.

Certain HSD3B1 gene types are linked to worse prostate cancer outcomes and affect treatment response and other health conditions.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Dual TCR Treg cells are common in various mouse tissues and show diverse characteristics.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

The protein CTCF is essential for skin development, maintaining hair follicles, and preventing inflammation.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

A genetic mutation in the CDH3 gene causes hair loss and vision problems in a young Saudi girl.