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Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

A specific gene mutation causes a severe skin disorder in a family.

A mutation in the KRTHB5 gene causes hair and nail issues.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

The ID2 gene can help distinguish between sensitizers and irritants in skin cells.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

N-WASP is essential for healthy skin and preventing inflammation.

Tianeptine may help prevent hair loss by promoting hair growth and reducing cell death.

ITK inhibitors may effectively treat alopecia areata.

DPP4, a molecule in skin, helps heal large wounds and regrow hair follicles when its levels are reduced.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

Targeting ATM could help manage heart cell enlargement due to pressure overload.

Hoxc13 regulates specific hair protein genes on mouse chromosome 16.

A rare gene variant causes hair and nail issues in a family.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Hoxc13 gene is essential for hair, nail, and papilla development.

The research showed that CRISPR/Cas9 can fix mutations causing a skin disease in stem cells, which then improved skin grafts in mice, but more work on safety and efficiency is needed.

Some Greek melanoma patients have gene mutations linked to increased cancer risk, a new color feature helps diagnose melanoma, the incidence of a skin condition in the Netherlands is rare, and a gene possibly affects male-pattern baldness.

Using CDK inhibitors on rats showed a reduction in chemotherapy-caused hair loss, but later experiments could not repeat these results.

PI3Kδ inhibition may effectively treat cutaneous chronic graft-versus-host disease.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Thymosin beta 4 protects cells from damage by blocking a harmful microRNA and boosting a protective gene.

AZD6244 treatment causes skin aging effects by depleting skin stem cells.

Deleting Crif1 in mouse skin disrupts skin balance and hair growth.

Edar and Eda proteins are crucial for proper tooth development.

High ornithine decarboxylase levels may lead to hair loss and cancer by increasing CK2 activity in the nucleus.

A new pair of mouse keratins, 65 kD and 48 kD, are found in specific skin areas and are linked to a unique skin differentiation type.

Too much keratin 16 in mice skin causes abnormal skin thickening and structure.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.