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Pigs without the Hairless gene showed skin and thymus changes, useful for studying human hair disorders.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

PP2Acα is essential for proper hair and skin development.

The study found that mouse sweat glands develop before birth, mature after birth, and have specific keratin patterns.

Hair loss in certain mice is linked to changes in keratin-related genes.

The hairless gene in Kunming mice is important for hair and skin, and shows genetic variations.

Mice with extra sheep genes had hair that fell out and regrew in cycles.

Keratin 6a is important for quick wound healing from hair follicles.

ING5 is crucial for stem cell maintenance and preventing certain cancers.

KF19418 promotes hair growth similarly to minoxidil but is not better in live mice.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

The scant hair in snthr-1Bao mice is likely caused by a deletion affecting the Plcd1 gene.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

The B2C promoter works in sheep cells but not in mouse embryos.

A specific gene region can control targeted and responsive gene expression in mice, useful for skin disorder treatments.

Researchers developed a mouse model that tracks hair growth using bioluminescence, improving accuracy in studying hair cycles.

Changing CDK4 levels affects the number of stem cells in mouse hair follicles.

Mice with human skin protein K8 had more skin problems and cancer.

The hairy ear mutation in mice is linked to changes in gene expression affecting hair growth.

Blocking a specific protein signal can make hair grow on mouse nipples.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Dab2 protein is crucial for hair follicle stem cell renewal and preventing early aging.

A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

The Gsdma3 gene is essential for normal hair development in mice.

Epidermal growth factor stops hair follicle formation in developing mouse skin.

Sgk3 is essential for normal hair follicle growth and maintenance.

The method successfully created stable transfection donor cells for goat hair follicle research.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.