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Androgen receptor inactivation delays skin cancer onset and reduces incidence in mice, with gender differences in susceptibility.

Nobiletin-loaded vesicles effectively treat skin cancer by restoring normal miRNA and antioxidant levels.

NKIRAS2 can suppress certain skin tumors but its effect on cancer varies with context and expression level.

Disrupting Bcl-xL in mice reduces skin cancer risk.

CXCR2 in skin cells promotes tumor growth.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

The method effectively induces skin cancer in mice for studying tumor development.

Deleting TNFα gene reduces skin cancer risk in certain mice.

LED light therapy at 863 nm wavelength can slow down skin tumor growth and reduce inflammation in mice.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

Grape seed extract is good for skin care, with antioxidant, anti-inflammatory, and anti-cancer effects.

The vitamin D receptor helps regulate skin and hair health independently of its usual vitamin D ligand.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

MCPIP1 in myeloid cells is important for skin cancer development and healthy hair growth.

Removing integrin α3β1 from hair stem cells lowers skin tumor growth by affecting CCN2 protein levels.

Meis1 is crucial for skin health and tumor development.

Vitamin D3 and its receptor help protect skin from UVB-related cancer.

Excessive putrescine causes hair loss in transgenic mice by disrupting hair follicle development.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

Older skin has higher cancer risk due to inflammation and stem cell issues.

sPLA2-IIA increases growth in hair follicle stem cells and cancer cells, suggesting it could be targeted for hair growth and cancer treatment.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Lgr5+ stem cells do not cause skin tumors.

A mouse gene mutation increases the risk of skin cancer.

Autophagy helps control skin inflammation and cancer responses and regulates hair growth by affecting stem cell activity.

UV exposure reduces Lgr6+ stem cells in mouse skin and they don't significantly contribute to skin cancer development.