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Nestin marks cells that can become a specific type of skin cell in hair follicles of both developing and adult mice.

Hair follicle-derived IL-7 and IL-15 are crucial for maintaining skin-resident memory T cells and could be targeted for treating skin diseases and lymphoma.

Vitamin D3 speeds up skin healing by helping skin stem cells grow and develop.

A new enzyme in rats may help regulate hair growth.

S100A3 protein is crucial for hair shaft formation in mice.

A new classification for trichothiodystrophy helps identify genetic causes and potential treatments.

CD8+ T cells drive alopecia areata, while regulatory T cells are protective.

Donor lymphocyte infusions effectively treated leukemia relapse but caused vitiligo and alopecia areata.

Bone marrow-derived cells contribute to skin tumors, suggesting new treatment targets for non-melanoma skin cancers.

A new genetic variant causes trichothiodystrophy in two brothers, but their mother may carry it without showing symptoms.

Higher levels of certain immune cells and proteins are linked to more severe lupus symptoms.

T-cells in alopecia areata scalp show abnormal regulation, leading to less inflammation.

A rare skin condition linked to leukemia improved with chemotherapy.

STAT3 signaling is important for healthy skin and hair follicles, and its disruption can lead to skin conditions like atopic dermatitis.

Netherton syndrome has two subtypes with shared immune traits but different allergic and immune responses, suggesting targeted treatments.

A specific type of immune cell plays a key role in causing alopecia areata and could be a target for treatment.

MG53 helps reduce skin damage caused by nitrogen mustard.

CD61 is important for mouse tooth cell growth and works through Lgr5.

Mouse hair follicle cells briefly grow during the early hair growth phase, showing that these cells are important for starting the hair cycle.

1,25-dihydroxyvitamin D3 helps melanocyte precursors from hair follicles grow and produce melanin, aiding vitiligo treatment understanding.

PDGF-BB helps young melanocytes grow but stops mature ones from growing, and it makes melanocytes more specialized.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Thorough evaluation and treatment are crucial for thyroid nodules.

MITF and WNT3A are key in Dun Mongolian horse pigmentation.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Telocytes have potential in therapy and tissue regeneration, but challenges in identification and cultivation remain.

Bone marrow and hair follicle cells help form skin tumors, suggesting new treatment targets.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.