Search

everythinglearnresearchcommunity

for

Search:↓

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

The mineralocorticoid receptor temporarily affects mouse skin development, but the glucocorticoid receptor has a more lasting impact.

A chromosomal change may cause ectodermal dysplasia and developmental issues in a child.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

A rare case of a transplant patient developing a skin condition linked to HPV-49.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Rat dermal papilla cells have unique properties and interact differently with their environment compared to other skin cells.

PSENEN gene mutations can cause both Dowling-Degos disease and acne inversa.

CD271 is crucial for maintaining healthy skin and preventing inflammation.

GLI2 increases follistatin production in human skin cells.

Scaffoldin helps form hard skin structures in chicken embryos.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

CD117 and platelet-derived growth factor receptor α may play a role in alopecia areata.

Dysplastic nevi have unique gene expressions, making them distinct from common melanocytic nevi.

Epigenetic mechanisms control skin development by regulating gene expression.

Different types of PPARγ are found in varying amounts in human skin and its parts, which could affect how skin treatments work.

A new PAX9 gene mutation causes missing teeth and hair problems, but not skin or nail issues.

Escin may help treat hair loss by boosting a specific cell growth pathway.

GLABRA2 represses root hair formation by inhibiting a specific gene.

MPA increases cancer spread by boosting Eph A2 activity.

The skin produces and responds to thyroid-stimulating hormone, which is controlled by other thyroid-related hormones.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Acid can block TRPV3 from outside the cell but boost its function from inside.

DS-2325a is safe and well-tolerated, supporting further development for Netherton Syndrome treatment.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vitamin D receptor is essential for hair growth signaling.