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Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

A new mutation in the mitochondrial DNA was found in a boy with MELAS, even though his family didn't show typical signs.

Hair root analysis can effectively detect somatic mosaicism in double cortex syndrome.

Scientists found a specific group of itch-sensing nerve cells in mice important for feeling itch but not for sensing heat or touch.

Harlequin mutant mice have hair loss due to low AIF protein levels and retroviral element activity.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

Nude mice are hairless due to a shared defect affecting both skin and thymus, not just thymic issues.

Muse cells from human bone marrow help reduce symptoms of atopic dermatitis in mice.

Activated protein C helps protect mice from radiation damage.

Giving a special protein to dogs with a certain genetic disease improved their symptoms but didn't help with hair growth.

The treatments stopped hair regrowth in mice.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

Two brothers from Bangladesh were diagnosed with X-linked adrenoleukodystrophy, showing neurological and adrenal symptoms.

The LMNA mutation affects skin structure even in asymptomatic carriers.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.

CD8+ T cells attack hair follicle stem cells, causing scarring and hair loss.

Genetic changes in mice help understand skin and hair disorders, aiding treatment development for acne and hair loss.

Retinoic acid affects skin and hair health by working with specific receptors, and its absence can lead to hair loss and skin changes.

A new mouse model for vitiligo helps study immune responses and potential treatments.

Activating β-catenin increases melanocytes and decreases Schwann cells.

A litter of cats had a hair condition similar to a mouse mutation, leading to hair loss and abnormal hair and skin.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Gender-affirming hormone therapy affects metabolism differently based on treatment type.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

MAA beads improved wound healing in male mice by activating the Shh pathway, but not in females.

The absence of functional sebaceous glands causes hair follicle destruction and scarring alopecia.

The Lanceolate hair-J mutation in mice mimics human hair disorders like Netherton's syndrome.