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Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

The PER3 rs772027021 SNP may cause mild skin pigmentation changes in a new subtype of dyschromatosis universalis hereditaria.

mTORC2 is crucial for healthy skin barrier by regulating lipids and filaggrin.

The mineralocorticoid receptor temporarily affects mouse skin development, but the glucocorticoid receptor has a more lasting impact.

Ectodin integrates BMP, SHH, and FGF signals in developing ectodermal organs.

Finasteride irreversibly affects human steroid 5α-reductase 2, providing insight into its catalytic mechanism and disease-related mutations.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

The human type II 5α-reductase gene, linked to certain male health conditions, has a specific structure and low similarity to other related genes.

Researchers found a genetic region that influences the number of coat layers in dogs.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

LGR5 mainly stays inside cells, moving to the trans-Golgi network, and this process is important for its role in cell signaling.

Delta-opioid receptors affect skin cell circadian rhythms, possibly impacting wound healing and cancer.

The VDR gene polymorphism does not affect BDNF levels in autoimmune thyroiditis and hypothyroidism patients.

The near-naked hairless mutation causes hair loss but is not due to a mutation in the hairless gene itself.

The workshop discussed the role of a protein called calreticulin in health and disease, its potential as a treatment target, and its possible use as a disease marker.

Gene therapy helped rats with a specific type of rickets grow hair without severe inflammation.

CRTH2 antagonists might be useful for treating many conditions because they play a role in immune and inflammation responses.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

ROR2 is essential for hair follicle stem cell renewal and maintenance.

Euph E and Euri A from Euphorbia neriifolia help control inflammation and immune response in cells.

Localized ROS production is essential for cell growth and movement in plants and animals.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

Edar signaling is crucial for proper hair follicle development and function.

LGR5 helps maintain corneal cell characteristics and prevents unwanted changes by controlling specific cell signaling pathways.

The CRABP I gene in cashmere goats is highly conserved but has unique features at specific amino sites.

A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

A protein called ectodysplasin-A2 increases a hair growth inhibitor in balding cells, which could be a target for hair loss treatment.