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Overexpression of sPLA2-IIA in mouse skin reduces hair stem cells and increases cell differentiation through JNK/c-Jun pathway activation.

Overexpressing follistatin in mice delays wound healing and reduces scar size.

Vitamin D receptor and mediator 1 are crucial for healthy skin and hair growth.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

The oncoprotein causes abnormal hair growth without increasing skin cancer risk.

Fish oil increased cell growth and macrophages in the skin but didn't affect COX-2 expression.

Rapamycin reduces skin cell growth and tumor development by affecting cell signaling in mice.

UVB exposure increases skin proteins for retinoic acid synthesis and shifts their location, possibly affecting skin repair.

UVB exposure affects retinoid metabolism, which is important for skin cancer progression and treatment.

CD18-deficient mice developed psoriasis-like skin disease, useful for studying inflammatory skin disorders.

Connexin mutations can cause various diseases like hearing loss and skin disorders.

Signaling factors and gene-driven cell adhesion are crucial for wound healing and embryo development.

The method effectively mimics shaving damage on skin for testing skincare products.

Vitamin D receptor helps prevent skin cancer and supports skin health.

The human keratin 6a gene's specific sequences trigger expression in skin layers after injury.

Activating Nrf2 in skin cells speeds up wound healing by increasing the growth of certain stem cells.

Iron is crucial for skin health, with specific proteins recycling it to support skin functions and prevent its release.

Chemotherapy drugs can cause abnormal growth and changes in healthy skin and gland cells.

The research found that specific stem cells maintain skin gland openings and that disrupting their activity can cause blockages or early cancer signs, indicating a need for targeted treatments.

Targeting JAK-STAT1 can reduce inflammation and promote hair growth in conditions linked to EGFR deficiency.

p63 may influence skin cancer development and cell differentiation.

YAP1 is important for skin regeneration and may affect skin disorder treatments.

Injecting follicular cells into skin can lead to the formation of new hair follicles.

Hidradenitis suppurativa tunnels have different microenvironments, suggesting targeted treatments could be more effective.

Hidradenitis suppurativa tunnels have different microenvironments, suggesting targeted treatments could be more effective.

Hidradenitis suppurativa tunnels have different microenvironments, suggesting targeted treatments could be more effective.

Hidradenitis suppurativa tunnels have different microenvironments, suggesting targeted treatments could be more effective.

Acute wounds heal well, but chronic wounds struggle due to ongoing inflammation and poor tissue repair.

Skin stem cells interacting with their environment is crucial for maintaining and regenerating skin and hair, and understanding this can help develop new treatments for skin and hair disorders.