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Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

The study developed a mouse model for Alopecia Areata that responds to treatment, useful for future research.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.

Suppressing ODC activity reduces tumor growth in hair follicles.

Controlling cellular changes can enable safe rejuvenation without cancer risk.

CRISPR-Cas9 successfully edited genes in Cashmere goats, affecting hair growth.

The method effectively induces skin cancer in mice for studying tumor development.

A nonviral method was developed to label and culture human hair follicle stem cells.

Blue laser light reduces energy in mouse skin cells and creates harmful oxygen compounds, possibly harming the cells.

Hair follicle stem cells might help treat traumatic brain injury.

Researchers found three types of melanocytes in developing mouse skin, each with different genes and locations.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

Two mouse mutations cause similar hair loss despite different skin changes.

Ablative fractional laser treatment nearly matches the gene reduction effects of topical vismodegib in skin cancer.

Zebrafish help understand genetic causes of skin pigment disorders like albinism.

Acute stress increases Y1 receptor gene expression in certain brain areas, but repeated stress does not.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Certain mouse strains develop a skin condition similar to a human hair loss disease due to genetic defects.

Transplanted rat hair follicles grew hair and had increased but not fully restored nerve connections in mice.

RXRa is crucial for Th2 immune cell development and may link nutrition to immune health.

Twist2 is essential for scarless skin healing and hair growth in mouse fetuses.

RT1640 treatment reverses gray hair and promotes hair growth in mice.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Abnormal keratin expression in mice causes severe oral issues, affecting feeding.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

Increasing SSAT makes skin more prone to cancer.