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PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

Some treatments can improve skin's defense against damage, but overuse may cause other skin problems.

Catalase in processed meats may cause cancer by emitting electromagnetic fields.

Gamma irradiation can stimulate hair growth by promoting new blood vessel formation.

Taxane chemotherapy can cause permanent hair loss by damaging hair follicles and altering their DNA.

Controlling cellular changes can enable safe rejuvenation without cancer risk.

TCDD changes skin cell growth and keratin production in mice.

A specific ATR gene mutation is linked to a hereditary oropharyngeal cancer syndrome.

Melatonin can help prevent and treat skin damage caused by gamma rays.

Keratin 10 end domains may increase skin cancer risk by reducing cell death.

Loss of TET2 increases the risk of skin and oral cancer.

A cancer patient died from a severe skin reaction after taking the drug cetuximab.

Dimethylarsinic acid speeds up skin tumor growth in certain mice.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

Loss of OGG1 increases skin inflammation and auto-antibodies in lupus.

Researchers created a rat model to study skin damage caused by radiation, which could help develop new treatments.

Excessive selenium intake can cause liver damage.

Vitamin D may help protect against nonmelanoma skin cancer.

Keratin 17 is important for skin's response to radiation, affecting many genes and cell division.

Mice with a specific BRCA1 mutation have a higher risk of tumors, especially in the uterus and ovaries.

Toxins can disrupt skin stem cell balance, causing skin overgrowth or ulceration.

Ionizing radiation damages human hair follicles by stopping cell growth, causing cell death, disrupting color, and increasing stress and damage markers.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Inducing survivin in normal tissues can protect against radiation damage.

Activating PPAR-γ signalling can protect hair follicle stem cells from damage caused by chemotherapy.

LGD1069 effectively prevents breast tumors in mice without toxicity.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.