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TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Glucosylceramides are essential for healthy skin and proper wound healing.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

Hair growth genes work better with more glucose due to changes in gene-regulating markers.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

Plasma protein binding significantly affects glucocorticosteroid concentration in blood, saliva, and hair.

A specific DNA region controls skin cell gene expression by working with certain proteins.

Alloxan diabetes, methylthiouracil, cortisone, and adrenaline affect how white mice hair follicles use glucose and cystine and their cell division.

Gasdermin A3 overexpression in skin causes inflammation and hair loss.

Targeting the HEDGEHOG-GLI1 pathway could help treat keloids.

Systemic glucocorticosteroids are a primary treatment for various skin conditions but require careful management due to potential side effects and relapses.

Overexpressing GLI-1 in mice skin can cause tumors like human basal cell carcinomas.

Overexpressing ornithine decarboxylase and v-Ha-ras in keratinocytes leads to invasiveness and malignancy.

The K15 promoter effectively targets stem cells in the hair follicle bulge.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Increasing mir-302 turns human hair cells into stem cells by changing gene regulation and demethylation.

Human adrenals and gonads have a unique enzyme for steroid hormone production.