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Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Modulating BMP activity changes the number, size, shape, and type of ectodermal organs.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Keratinocytes can reverse the effects of the GNAQ oncogene, inhibiting melanoma cell growth.

The gene Endothelin 3 makes mice's fur darker by increasing pigment cells and pigment levels.

Lhx2 is a crucial regulator of the Sonic Hedgehog signaling in early mouse retinal development.

Scientists created cell lines to study a genetic skin disorder using CRISPR technology.

Dkk4 is necessary for the initial development and arrangement of hair follicles.

Minoxidil helps restore thymus size in 22q11.2 deletion syndrome.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Iron deficiency causes hair loss by affecting hair differentiation and cycling.

Hereditary factors cause hair loss in mice by affecting skin and hair follicle structure.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

The Oat mouse model shows mild retinal degeneration, useful for testing treatments.

Transplanted rat hair follicles grew hair and had increased but not fully restored nerve connections in mice.

WWOX deficiency in mice causes skin and fat tissue problems due to disrupted cell survival signals.

Nelfb is essential for dermal fat development and survival.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Mice with more Flightless I protein grew back their claws better after amputation.

The new rodent model successfully mimics non-lean human PCOS symptoms.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Finasteride treatment changes Cx43 in rat testes, possibly causing fertility issues.

Lack of vitamin D receptor increases skin tumor risk by boosting hedgehog signaling.

Scientists identified and cloned specific keratin proteins in mouse hair.

Hairless HRS/J mice resist Bacillus anthracis skin infections due to high numbers of immune cells, not because they lack hair follicles.

A zinc-deficient diet stunted growth and affected organs in mice, with C57BL/6J mice showing more severe symptoms.

The nude gene causes skin cell overgrowth and improper development, leading to hair and urinary issues.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.