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The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

The PP2A-B55α protein is essential for brain and skin development in embryos.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Expressing the human vitamin D receptor in skin cells prevents hair loss in certain mice.

EGFR is essential for normal hair development and follicle differentiation.

A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

GFRα2 is essential for controlling neuron size but not for target innervation in certain sensory neurons.

LGD1069 effectively prevents breast tumors in mice without toxicity.

Removing myelin protein zero-like 3 in mice leads to better metabolism and resistance to obesity.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Defective mitochondrial DNA replication causes aging symptoms and increased oxidative damage in mice.

The treatments stopped hair regrowth in mice.

Deleting part of a gene in mice causes wavy hair and high pup loss.

The antibody 005-C04 blocks prolactin receptors, causing reversible infertility, impaired lactation, and hair regrowth in female mice.

The model using human skin on mice helps study human sebaceous glands.

Gamma-ray exposure improves genome editing efficiency in mice using the i-GONAD method.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

The naked mutation in mice causes hair loss and helps identify keratin genes.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

Deleting the Trf1 protein in mice is safe and may help prevent cancer without major side effects.

The mouse model helps study and develop treatments for radiation-induced saliva reduction.

Lack of KSR1 stops certain skin tumors in mice.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

Mice without the Sp6 gene have problems developing several body parts, including hair, teeth, limbs, and lungs.

Mice with a changed Hr gene lose and regrow hair due to changes in the gene's activity.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.