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Toluene diisocyanate exposure can cause immune sensitization by interacting with proteins in hair follicles and sebaceous glands.

Dermal IgA deposition without symptoms is rare in Dermatitis herpetiformis risk groups.

An enzyme other than TNAP might be responsible for vitamin B-6 metabolism in the skin.

Two specific hair keratin genes are active during hair growth and decline as hair transitions to rest.

A gene mutation in mice causes skin defects and early death.

Blocking CXCR4 may help treat hidradenitis suppurativa.

Tiger tail banding and hair abnormalities are reliable indicators for diagnosing trichothiodystrophy.

CAP1/Prss8 does not activate PAR2 or inhibit PN-1.

A defective protein in progeria causes cell death and atherosclerosis, but a treatment targeting cell stress may reduce these effects.

A small change in the TRPS1 gene leads to a less severe form of a syndrome affecting hair, nose, and finger development.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, which could be a new target for treating hair loss and other skin conditions driven by testosterone.

A new mutation in the ST14 gene causes a rare skin and hair disorder in a specific family.