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Scientists found new and known long non-coding RNAs in mouse hair follicle stem cells that may be important for stem cell function and could be targets for cancer treatment.

PBX1 helps hair stem cells grow and change by turning on certain cell signals and preventing cell death, which may be useful for hair regrowth treatments.

HPV8 E6 gene causes growth of certain skin stem cells.

CDKN2AIP gene is less active in nevus sebaceous, affecting related RNA networks.

EZH2 is crucial for uterine gland development and female fertility.

Polyamines help fix DNA damage accurately in cells.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

LHX2, with other markers, can identify hair placodes in rats.

A new neural network helps identify key regulators in cell changes, aiding in understanding diseases and finding new treatments.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

Keratins K6 and K16 are expressed more freely in regenerating mouse skin than K1 and K10.

Hairless protein and putrescine regulate each other, affecting hair growth and skin balance.

The MtAnn3 gene affects root hair growth and is influenced by cytokinin.

Overactive Stat3 in mouse skin causes hair loss and cell structure damage.

Mouse keratin 6 isoforms have different expression patterns in various tissues.

KLF4 is important for keeping hair follicle stem cells inactive.

Overexpressing ATF3 in mice's epithelial cells may lead to oral cancer.

Certain genetic variants in ERN1, TACR3, and SPPL2C are linked to when Alzheimer's disease starts.

Higher childhood DHEAS levels are linked to advanced puberty and DNA changes.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

Protein Kinase Cε increases skin sensitivity to UV damage and skin cancer risk.

Altering SSAT affects fat metabolism and body fat in mice.

Mutations in hKAP1 genes may cause hereditary hair disorders.

Zeb2 is crucial for nerve repair by controlling Schwann cell function.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

Loss of Dnmt3a and Dnmt3b increases aggressive skin tumors by affecting PPAR-γ.