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Scientists made a mouse that shows how a specific protein in the skin changes and affects hair growth and shape.

The protein Par3 is crucial for healthy skin, affecting the skin barrier, cell differentiation, and stem cell maintenance.

ILK and ELMO2 help cells move and stick together, important for wound healing and hair growth.

Two mouse mutants have defective hair cuticle cross-linking.

Basement membrane supports fibroblast aggregation, aiding hair follicle development.

Nephronectin helps attach muscle cells to hair follicles.

RPGRIP1L helps skin cells stick together by blocking PKCβII, which can prevent skin blistering like in pemphigus.

The cornified envelope is crucial for skin's barrier function and involves key proteins and genetic factors.

Skin cells from people with Epidermolysis Bullosa Simplex have abnormally placed and less active mitochondria.

Hair cuticles remain stable and resilient under stress due to strong protein content and crosslinking.

Human TMEM2 does not break down hyaluronan but helps control its metabolism.

Meis1 is crucial for skin health and tumor development.

Corneal health relies on cell migration and cooperation with the lacrimal gland, not Bmi1+ cells, and Eda gene mutations can cause dry eye disease.

A specific group of skin stem cells was found to help maintain hair follicle cells.

Dermal EZH2 controls skin cell growth and differentiation in mice.

Ceramide synthase 4 is essential for maintaining skin barrier health.

Hox proteins help maintain keratinocyte identity by regulating miRNA expression.

Sostdc1 controls the size and number of hair and mammary gland structures.

A specific gene mutation causes sparse, brittle hair in a family.

Newly born mesenchymal cells quickly spread out in response to tissue tension during early development.

Some mutant mice have hair with abnormal cross-linking, mainly in the cuticle, not affecting other hair parts.

Disulfide bonds in keratin fibers break more easily under stress, especially when wet, affecting fiber strength.

H3K9me3 is crucial for proper skin development and structure.

Sdr16c5 and Sdr16c6 genes regulate a key point in lipid production that affects eye and skin gland function.

Hair follicles in the back of the rosette fancy mouse have reversed orientations due to a gene mutation.

Proper niche formation in Drosophila requires Slit-Robo signaling for cell migration.

The tumor suppressor BRCA2 helps in cell division by bringing key proteins to the area where cells split.