Search

everythinglearnresearchcommunity

for

Search:↓

Immortalized hair follicle cells could be useful for regenerative medicine and treating inflammation and oxidative stress.

Androgens may cause hair loss by increasing TGF-beta1 from scalp cells, which inhibits hair cell growth.

Human dermal fibroblasts are the main cells targeted by a virus that can cause a deadly skin cancer, and a certain inhibitor can effectively block this infection.

Moracin M helps hair grow by activating specific pathways and improving blood vessel formation.

Pro-Ile could be used in cosmetics to improve skin, hair growth, and healing.

Manipulating EGFR signaling may help treat hair loss and promote hair growth.

Botulinum toxin type A and platelet-rich plasma together boost hair growth by activating specific growth pathways.

Inhibiting the PI3K/Akt pathway may help prevent radiation-induced liver injury.

EGFRi/MEKi treatments cause hair follicles to lose some immune protection, leading to inflammation.

Activating certain hair follicle cells could prevent hair loss from cancer treatments.

EGF and FGF signaling stops hair follicle development in mice.

Parthenolide promotes hair growth in mice and may influence pathways related to male pattern baldness.

Genetic mutation and carcinogen treatment are both needed for skin cancer to develop in these specific mice.

Minor skin injuries can boost hair growth by increasing TNF-α.

Delivering IGF-1 with PLGA microspheres improves stem cell regeneration for tissues.

PRSS35 enzyme may help start skin tumors and could be a target for cancer treatment.

Overexpression of E2F1 can lead to skin tumors and disrupt hair growth.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Human liver enzyme DHEA ST helps process minoxidil.

VEGF can boost hair growth in mice with scleroderma.

Sorafenib after liver surgery may increase survival for some liver cancer patients.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

People respond differently to hair loss treatment with PRP because of individual differences in growth factors from platelets.

IGF-I promotes hair growth by keeping hair follicles in the growth phase longer.

Human foreskin fibroblast–conditioned medium improved hair growth in androgenetic alopecia patients.

Advancements in tissue engineering show promise for hair follicle regeneration to treat hair loss.

Inhibiting glycogen metabolism can promote hair growth.

IGF2BP3 gene is up-regulated in keloid patients, suggesting potential targets for treatment.

Shen-Min, a hair growth supplement, likely caused acute hepatitis in a woman, improving after she stopped taking it.