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Hair follicle stem cell transplants can reverse liver cirrhosis by blocking harmful cell activation.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

The Bsk mutation doesn't involve keratin gene recombination and its cause is unknown.

Zebrafish help understand genetic causes of skin pigment disorders like albinism.

The we/we wal/wal mice have defects in hair growth and skin layer formation, causing hair loss, useful for understanding alopecia.

Keratin 16 delays skin maturation and affects skin and hair development in mice.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

Hair loss in certain mice is linked to changes in keratin-related genes.

Removing both KLK5 and KLK7 proteins can prevent death and skin issues in Netherton syndrome.

Removing MCPIP1 from myeloid cells in mice leads to hair loss and prevents skin tumors but causes pigmented spots.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Lack of KSR1 stops certain skin tumors in mice.

Hairless mice lose hair by 3-4 weeks, develop thicker, folded skin, and show pigmentation differences.

Disrupting the Hars2 gene in mice causes hearing loss due to mitochondrial problems and hair cell damage.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

The HCR gene contributes to psoriasis risk.

Human cathepsin V can replace mouse cathepsin L to maintain normal skin and hair in mice.

Whn is essential for hair growth, and its malfunction causes hair loss.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

The mouse model could be useful for baldness research and testing treatments like testosterone, cyproterone acetate, and minoxidil.