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A gene variant increases the risk of a type of hair loss by affecting hair protein production.

The study found nine new hair protein genes in human hair follicles.

Genes related to keratin, skin cell differentiation, and immune functions are key in hedgehog skin and spine development.

Autophagy is important for determining the protein makeup of hair.

NFATc1 controls hair stem cell activity, affecting hair growth and could be a target for hair loss treatments.

Fgf9 helps create new hair follicles after injury through skin T cells.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

Targeting protein S-palmitoylation could lead to new skin disease treatments.

ZDHHC13 is important for normal liver function and metabolism, affecting mitochondrial activity.

The HR protein helps hair grow by blocking a hair growth inhibitor, aiding in hair follicle regeneration.

A cluster of sulfur-rich hair protein genes was found on chromosome 17.

Three specific mutations in the LIPH gene can cause hair loss by damaging the protein's structure and function.

The hairless (hr) gene is essential for normal hair follicle function and its mutation leads to hair loss.

The Notch signaling pathway helps in mouse hair development through a noncanonical mechanism that does not rely on RBPj or transcription.

Researchers mapped and categorized specific keratin-associated protein genes on human chromosome 21q22.1.

HR protein causes abnormal hair cycles by increasing Tgf-β2 and reducing miR-31.

Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

Blocking CXCR4 may help treat hidradenitis suppurativa.

PIEZO1 helps keep hair follicle stem cells inactive, affecting hair growth.

Smad1 and Smad5 are essential for hair follicle development and stem cell sleepiness.

Hair follicle stem cells change states with age, affecting hair growth and aging.

Mutations in the DSG4 gene cause fragile, sparse hair in humans, mice, and rats.

Mutations in the FOXN1 gene cause severe immune issues but don't affect hair and nails.

Keratin 15 expression in skin cells is regulated by two mechanisms involving PKC/AP-1 and FOXM1.

The GPRC6A-Duox1 axis helps control hair growth and loss by affecting hydrogen peroxide production.

SOX11 and SOX4 help skin cells act like embryonic cells to heal wounds in mice.

Dermal fibroblasts have adjustable roles in wound healing, with specific cells promoting regeneration or scar formation.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

R-spondin2 may help treat hair loss, gene differences could explain baldness, a peptide's regulation is linked to psoriasis, B-defensin gene copies may affect a skin condition's risk and severity, and potential markers and targets for alopecia areata were identified.