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Melanocyte-associated antigens may play a key role in alopecia areata and could be targets for new treatments.

A specific mutation in PA-PLA1α causes abnormal hair growth.

The Hairless gene is crucial for healthy skin and hair growth.

The human type II 5α-reductase gene has a specific structure important for understanding certain medical conditions.

Higher PD-1 levels mean fewer CD8+ T cells in alopecia areata hair follicles.

The study found two new mutations in a Chinese patient with severe biotinidase deficiency.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

miR-200c-3p could help diagnose and treat alopecia areata.

The mK6irs1/Krt2-6g gene likely causes wavy hair in mice.

NKG2D+CD4+ T cells are higher in alopecia areata patients and may be involved in the disease.

Genetic testing is crucial for diagnosing and treating acrodermatitis enteropathica effectively.

A faulty KLHL24 gene leads to hair loss by damaging hair follicle stem cells.

PDCD4 protein levels, affected by genetics and breastfeeding, may predict breast cancer treatment response.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

Two specific SNPs in the TRPS1 gene cause excessive hair growth by altering the protein's structure.

SLC24A5 shows a clear selective sweep, but no link to UV radiation intensity.

Genetic changes in specific proteins contribute to hair loss in some women of African descent.

New mutations in the DSG4 gene cause a rare hair condition.

A specific gene mutation causes a rare hair loss condition in a Chinese patient.

Injecting recipient splenocytes into donors' thymus can prevent graft-versus-host disease.

The Fas/FasL pathway may play a role in alopecia areata.

Certain gene variations in EGF and EGFR may increase the risk of alopecia areata in Koreans.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

A genetic duplication on chromosome 5 was linked to a woman's unique combination of medical conditions.

Anti-DFS70 antibodies can help identify nonsystemic autoimmune conditions in SLE patients.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Blocking CCR5 can prevent and improve hair loss in alopecia areata.

A mutation in the human hairless gene causes alopecia universalis.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.