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A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

Low Vitamin D receptor levels in breast cancer are linked to worse outcomes and more bone metastases, and could be a marker for prognosis.

Mutations in the vitamin D receptor cause hereditary vitamin D-resistant rickets, leading to poor bone health and requiring high calcium doses for treatment.

A new mutation in the HR gene is linked to a rare form of hair loss with limb deformities.

A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

A protein called PCBP2 controls the production of a hair growth protein by interacting with its genetic message and is linked to hair loss when this control is disrupted.

The ABHRS held a test in Capri, Italy.

Most Hairless gene mutations reduce its ability to work with the Vitamin D Receptor, which might explain a certain type of hair loss.

A mutation in the HR gene causes a rare form of irreversible hair loss in two Kashmiri families. Whole exome sequencing is effective for finding such mutations.

Intravenous calcium therapy is a safe and effective treatment for a rare type of rickets.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

A new method was developed to accurately detect and measure 47 different drug ingredients in various products.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

Vogt-Koyanagi-Harada disease affects vision and skin, mainly in people with darker skin, and is treated with steroids and immunosuppressants.

The HR protein helps hair grow by blocking a hair growth inhibitor, aiding in hair follicle regeneration.

A vitamin D receptor mutation causes rickets and affects immune responses.

Vogt-Koyanagi-Harada disease can develop during interferon therapy for chronic hepatitis C.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

The vitamin D receptor is involved in multiple body functions beyond calcium regulation, including immune response and rapid reactions not related to gene activity.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

A specific gene mutation causes vitamin D-resistant rickets and hair loss.

Medical Hair Restoration added a non-surgical hair therapy program to help people with early hair loss.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Vitamin D receptor binds similarly to natural and synthetic ligands, affecting gene regulation.

Researchers created a mouse model of a type of rickets that does not cause hair loss.