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Hidradenitis suppurativa may be a type of autoinflammatory skin disease linked to gene mutations and immune system issues.

A new mouse hair mutation, called hague, is semidominant and unstable, but the exact cause is unknown.

Faulty LEF1 activation causes faster skin cell differentiation in premature aging syndrome.

Increased HIF-1α is linked to the inflammation and severity of hidradenitis suppurativa, suggesting treatments that lower HIF-1α could help.

Hedgehog signaling controls hair follicle development and can affect skin cancer growth.

Impaired LEF1 activation speeds up skin cell development in Hutchinson-Gilford Progeria Syndrome.

Notch1 signaling is impaired in hidradenitis suppurativa, affecting skin and hair cells.

A new genetic mutation linked to CARASIL syndrome and small artery disease was found in a Chinese family.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

Blocking CXCR4 may help treat hidradenitis suppurativa.

Sex and race affect immune responses and treatment outcomes in Hidradenitis suppurativa.

Using SNP array testing helped quickly find the gene causing Woodhouse-Sakati syndrome in two related individuals.

The mutation causing Hutchinson-Gilford progeria syndrome leads to severe skin problems and early death in mice.

Hidradenitis suppurativa might be a type of autoinflammatory skin disease.

A new mutation in the ST14 gene broadens the understanding of ichthyosis-hypotrichosis syndrome.

A woman with a long-term skin condition developed a serious skin cancer that led to her death.

Skin organoids with NCSTN mutation show changes in hair follicle development and higher inflammation, key features of Hidradenitis Suppurativa.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.

The mutant HR bmh protein mis-localizes in cells, affecting skin and hair development.

Human placenta extract reduces inflammation and symptoms in atopic dermatitis.

Certain proteins are significantly increased in the skin of people with hidradenitis suppurativa.

A mutation in the hairless gene speeds up severe itchy skin in mice on a special diet.

H1 increases risk for neurodegenerative diseases, while H2 offers protection but is linked to other disorders.

HLD10 can include increased body hair and Mongolian spots.

A new protein linked to hair strength was identified, aiding in understanding brittle hair conditions.

A mutation in the HR gene causes a rare form of irreversible hair loss in two Kashmiri families. Whole exome sequencing is effective for finding such mutations.

Hidradenitis suppurativa is a chronic skin disorder linked to genetics, lifestyle, and immune issues, treatable with medication and sometimes surgery.

Hidradenitis suppurativa is a chronic skin disorder linked to genetics, lifestyle, and immune issues, treatable with medications and surgery.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.