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Apoptosis in hair follicles spreads through cell death signals, with stem cells slowing the process.

Oxidative stress plays a significant role in vitiligo, and both skin and non-skin cells may be involved.

Melanoma risk tools need improvement, a gene mutation causes a hair disorder that might be treated by managing cell stress, a potential therapy for a skin-ear disorder involves blocking cell channels, skin wrinkling may indicate lung aging regardless of smoking, and oxidative stress might contribute to common baldness.

Dormant melanoma cells in mice interact minimally with memory T cells due to a suppressive tumor environment.

Androgens increase pigmentation in young hamsters, but estrogens can reverse this effect.

MITF+ melanoma patients are more likely to have multiple melanomas and unique skin patterns.

Notch/RBP-J signaling is crucial for proper placement and timing of melanocyte development in hair follicles.

Mitf plays a key role in melanoma progression and is linked to disease stage.

Hydrogen peroxide is a major risk factor for melanoma in swimmers, more than UV light.

PDGF-BB helps young melanocytes grow but stops mature ones from growing, and it makes melanocytes more specialized.

A clinically suspected melanoma appeared benign under the microscope but was confirmed by specific tests and a rare mutation.

The new assay can track and measure melanosome transfer between skin cells, confirming filopodia's role in this process.

The protein p21 is more abundant in normal skin cells than in melanoma cells and may help protect against melanoma, with UVB light affecting its levels.

Exosomes from umbilical cord stem cells help protect hair color and health under stress.

Bone marrow-derived cells contribute to skin tumors, suggesting new treatment targets for non-melanoma skin cancers.

Researchers successfully grew horse skin cells that produce pigment from hair follicle samples.

UV light might cause excessive hair growth by increasing PGE2 in the skin.

Feather pigment patterns form through melanocyte arrangement and simple regulatory mechanisms.

c-Myc activation in mouse skin increases sebaceous gland growth and affects hair follicle development.

Sox2 controls hair color by affecting pigment production in hair follicles.

BMP signaling controls hair growth and skin color.

Chronic stress can reduce skin pigmentation.

BMP4 helps stem cells turn into pigment-producing cells, affecting hair color and growth.

Misusing topical corticosteroids can cause skin darkening and other side effects.

Hair color is influenced by genetics and can indicate certain health conditions.

PCE-DP brightens skin and promotes hair growth by enhancing cell growth and reducing melanin uptake.

New findings suggest targeting IL-23 could treat psoriasis, skin cells can adapt to new roles, direct conversion of skin cells to blood cells may aid cell therapy, removing certain tumor cells could boost cancer immunotherapy, and melanoma may have many tumorigenic cells, not just cancer stem cells.

A rare pigmented skin tumor called melanotrichoblastoma was diagnosed in a 51-year-old woman.

White hair follicles in vitiligo have fewer and less functional melanocytes.