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Complete heart block in lupus may be linked to microvascular issues and anti-PM-Scl 100 antibodies, not just traditional markers.

Adalimumab may cause severe hair loss in some patients.

Nonimmunogenic forms of keratins K71 and K31 can delay and prevent alopecia areata.

Higher levels of certain DNAs in blood may indicate hair follicle damage in alopecia areata patients.

A man developed myasthenia gravis after alopecia areata, and treatment improved his symptoms and stopped hair loss.

A woman with type 2 diabetes developed alopecia areata, suggesting a link between the two autoimmune conditions.

Alopecia areata involves persistent gene abnormalities and immune activity, even in regrown hair, suggesting a risk of relapse.

Baricitinib is a promising treatment for alopecia areata.

Hidradenitis suppurativa might be a type of autoinflammatory skin disease.

The Italian guidelines offer advice for diagnosing and treating alopecia areata based on expert opinion and limited clinical trial evidence.

Vδ1+ T-cells in the skin contribute to hair loss in alopecia areata and could be targeted for treatment.

Alopecia areata involves immune response and gene changes affecting hair loss.

Hidradenitis suppurativa is linked to autoinflammation and immune system issues.

New treatments for alopecia areata show promise but need ongoing use and have infection risks.

Tofacitinib effectively treated a woman's complex autoimmune and autoinflammatory disorders.

New treatments for alopecia areata focus on targeting immune responses for better results.

Blood tests can help understand the genetic differences in people with alopecia areata, including how severe it is and if it's inherited.

Protein microarrays are highly sensitive tools useful for disease diagnosis and studying proteins.

Alopecia areata involves immune issues, inflammation, and nutrient deficiencies.

JAK1 inhibitor may effectively treat alopecia areata when TNF-α inhibitors and corticosteroids fail.

Mycophenolate mofetil can effectively treat autoimmune hepatitis when standard drugs fail.

A woman with undifferentiated connective tissue disease was diagnosed with renal amyloidosis and carpal tunnel syndrome.

Serum granulysin levels can indicate the activity and prognosis of alopecia areata.

Certain immune-related proteins are higher in people with alopecia and their healthy relatives, hinting at a genetic link.

Alopecia areata patients have fewer protective regulatory B cells, which may contribute to the disease.

A humanized CXCL12 antibody may delay and treat alopecia areata by altering the immune response.

ILC1-like cells can cause alopecia areata by affecting hair follicles.

Prior infections reduced alopecia areata development in mice.

Alopecia areata severity is linked to increased TH1 and TH2 activity.