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Certain cytokines may cause or be affected by immune skin diseases, suggesting possible new treatments.

Psoriasis is linked to a higher risk of autoimmune diseases and other health conditions.

Innate lymphoid cells type 1 may contribute to alopecia areata.

Super-enhancers control CD25 expression in specific cell types, affecting immune function.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

Psoriasis patients on biologic therapy have a weaker immune response to COVID-19 vaccination than healthy people.

AIRE protein, defective in APECED patients, is found in skin and hair cells and interacts with cytokeratin 17.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

Plasmacytoid dendritic cells, which overproduce IFN-α, may play a crucial role in starting alopecia areata, an autoimmune disease causing hair loss.

PLAU and SerpinB2 affect cell death differently in various forms of leprosy and could be targets for new treatments.

Collagen XVII is crucial for skin cell growth and nail health.

ASLAN004 was safe and well-tolerated, supporting further development for treating certain diseases.

Laminin-511 is involved in psoriasis development and can be regulated by cannabinoid receptor type 1.

Certain genetic variations in IL18 may increase the risk of alopecia areata in Koreans.

Atopy and altered T cell functions contribute to Alopecia Areata.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Higher interleukin-15 levels are linked to more severe alopecia areata, but not to age or gender.

ILC1-like cells may contribute to hair loss in alopecia areata.

Collagen XVII is important for cell functions and its absence can worsen cancer outcomes.

Hair follicles produce IL-7, which is essential for certain skin lymphoma cells to survive.

Increasing type 17 collagen reduces aging signs in skin cells caused by UV light.

Innate lymphoid cells type 1 may contribute to alopecia areata by damaging hair follicles.

IL-15 and TNF-α levels are higher in alopecia areata patients, especially in alopecia totalis.

CD27 and IL-35 can help diagnose alopecia areata linked to bacterial infections.

IL-1α levels are higher in alopecia areata patients, suggesting a role in the disease.

A fragment of human type XVII collagen shows great potential for skin health and wound healing.

Collagen VII helps skin heal and stay strong, sirolimus may lower skin cancer risk in kidney transplant patients, high-molecular-mass hyaluronan helps naked mole rats resist cancer, dermal γδ T cells aid in hair growth in rodents, and overexpression of IL-33 in mouse skin causes itchiness, offering a model for studying allergic inflammation treatments.

Targeting MIG and MCP-1 may help treat inflammation in alopecia areata.