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Double-stranded RNA causes inflammation in hair follicle cells, which may help understand and treat alopecia areata.

ILC1-like cells can independently cause alopecia areata.

IL-15 and TNF-α levels are higher in alopecia areata patients, especially in alopecia totalis.

Certain genetic variations in IL18 may increase the risk of alopecia areata in Koreans.

PLAU and SerpinB2 affect cell death differently in various forms of leprosy and could be targets for new treatments.

Psoriasis may be chronic because it lacks certain immune system controls that prevent overreaction.

ILC1-like cells may contribute to hair loss in alopecia areata and could be new treatment targets.

Treatment with biologic agents can significantly improve psoriasis symptoms, and blood biomarkers could potentially predict individual patient's response to treatment.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

T cells affect skin cell genes in inflammatory diseases, and therapy can normalize these changes.

A new genetic region, 17q21.31, is linked to higher ovarian cancer risk.

IL-1α levels are higher in alopecia areata patients, suggesting a role in the disease.

Enhancing Tregs can protect against alopecia areata.

Inflammation in rheumatoid arthritis increases steroid activation in cells, reducing inflammation.

IL-33 is linked to hair follicle damage in psoriasis and could be a treatment target for hair loss in this condition.

IL-1 receptor absence in mice leads to skin cysts and changes in immune response after UVB exposure.

Atopy and altered T cell functions contribute to Alopecia Areata.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

Keratin 17 is crucial for cell growth in wound healing by aiding protein synthesis.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

ILC1-like cells can cause alopecia areata by attacking hair follicles.

PX-12 may help treat psoriasis by blocking inflammation and cell death.

ILC1-like cells may contribute to hair loss in alopecia areata.

Higher CD70 and CD27 gene expression in alopecia areata lesions predicts disease severity and activity.

ILC1-like cells can independently cause alopecia areata by affecting hair follicles.

Memory regulatory T cells need IL-7, not IL-2, to stay in peripheral tissues.

Keratin 17 is important for hair and nail structure and affects pachyonychia congenita symptoms.

Removing UBE2N from skin cells causes inflammation and immune response, which can be lessened with specific inhibitors.

Increased Treg cells and IL-10 may help quick recovery in acute diffuse and total alopecia.