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TIP39 and PTH2R help control calcium levels and skin cell development.

Blocking JAK1 or JAK3 helps reverse hair loss in a mouse model of alopecia areata.

miR-218-5p helps skin and hair growth by targeting SFRP2 and activating a specific signaling pathway.

Nelfb is crucial for forming skin fat tissue by regulating genes needed for fat cell development.

The choice between belimumab and anifrolumab depends on the specific symptoms of the patient's lupus.

Tet1/2/3 enzymes affect hair follicle cell development by influencing BMP signaling.

CCL5 is important for the hair growth potential of human dermal papilla cells.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

Expanding CD4+ Tregs can stop hair loss in alopecia areata.

Lnc056 helps hair follicle stem cells grow by increasing TRIP6 expression.

Changes in certain RNA and protein levels may contribute to alopecia areata and could be treatment targets.

CCL 27 levels are similar in people with and without alopecia areata.

The Hr protein binds to DNA, interacts with p53, and affects cell cycle genes.

Hairless protein can both repress and activate vitamin D receptor functions, affecting gene regulation.

Ptch2 plays a key role in controlling stem cell function and the ability to regenerate after birth.

The enzyme DHHC13 is essential for healthy hair and skin, and its deficiency leads to hair loss and skin problems.

ASLNC168501 can help treat hair loss by restoring hair follicle stem cell function.

DNMT1 helps turn hair follicle stem cells into fat cells by blocking a specific microRNA.

A substance called poly(I:C) increases a protein called carbonic anhydrase II in skin cells, which might help with skin defense and healing.

Ixekizumab may be an effective first treatment for a rare skin condition causing hair loss.

Telitacicept effectively improved hair regrowth in a woman with lupus and alopecia.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

Dimethyl fumarate speeds up wound healing in IL-36Ra deficient mice by reducing NET formation and oxidative stress.

Sp6 promotes tooth development by reducing follistatin levels.

Different enzymes are active in different parts of developing mouse organs.

Laminin 332 is essential for normal skin cell behavior and structure.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

Keratin 17 is linked to various diseases, including cancer and skin conditions, and may be a target for diagnosis and treatment.